Mitochondrial dysfunction in a family with psychosis and chronic fatigue syndrome.
Mitochondrial dysfunction in a family with psychosis and chronic fatigue syndrome. Read More »
Torrell H et al. (2017) Mitochondrial dysfunction in a family […]
Research Papers
Exosome-Associated Mitochondrial DNA from Patients with ME/CFS Stimulates Human Cultured Microglia to Release IL-1β.
Tsilioni I et al . (2022) Exosome-Associated Mitochondrial DNA from
Increased Resting Energy Expenditure in the Chronic Fatigue Syndrome.
Increased Resting Energy Expenditure in the Chronic Fatigue Syndrome. Read More »
Watson WS et al. (1998) Increased Resting Energy Expenditure in
Role of mitochondria, oxidative stress and the response to antioxidants in myalgic encephalomyelitis/chronic fatigue syndrome: A possible approach to SARS-CoV-2 ‘long-haulers’?
Wood E et al. (2020) Role of mitochondria, oxidative stress
Excessive intracellular acidosis of skeletal muscle on exercise in a patient with a post-viral exhaustion/fatigue syndrome.
Arnold DL et al. (1984) Excessive intracellular acidosis of skeletal
Prevalence of Aspergillus-Derived Mycotoxins (Ochratoxin, Aflatoxin, and Gliotoxin) and Their Distribution in the Urinalysis of ME/CFS Patients.
Wu TY et al. (2022) Prevalence of Aspergillus-Derived Mycotoxins (Ochratoxin,
Oxidative stress caused by a dysregulated Wnt/beta-catenin signalling pathway is involved in abnormal placenta formation in pregnant mice with chronic fatigue syndrome.
Zhao H et al. (2020) Oxidative stress caused by a
Mitochondrial myopathy presenting as fibromyalgia: a case report.
Mitochondrial myopathy presenting as fibromyalgia: a case report. Read More »
Abdullah M et al. (2012) Mitochondrial myopathy presenting as fibromyalgia:
Mitochondria and immunity in chronic fatigue syndrome.
Mitochondria and immunity in chronic fatigue syndrome. Read More »
Anderson G and Maes M (2021) Mitochondria and immunity in
Lymphoblastoid Cell Lines as Models to Study Mitochondrial Function in Neurological Disorders.
Annesley SJ and Fisher PR (2021) Lymphoblastoid Cell Lines as
A View Into the Mechanisms Underlying TORC1, AMPK, and Complex V Activity in Myalgic Encephalomyelitis Using the Dictyostelium discoideum Model.
Arnold B (2022) A View Into the Mechanisms Underlying TORC1,
Mitochondrial abnormalities in the post-viral fatigue syndrome.
Mitochondrial abnormalities in the post-viral fatigue syndrome. Read More »
Behan WMH et al. (1991) Mitochondrial abnormalities in the post-viral
Suggested Pathology of Systemic Exertion Intolerance Disease: Impairment of the E3 Subunit or Crossover of Swinging Arms of the E2 Subunit of the Pyruvate Dehydrogenase Complex Decreases Regeneration of Cofactor Dihydrolipoic Acid of the E2 Subunit.
Bohne V and Bohne O (2019) Suggested Pathology of Systemic
Abnormalities of AMPK activation and glucose uptake in cultured skeletal muscle cells from individuals with chronic fatigue syndrome.
Brown AE et al. (2015) Abnormalities of AMPK activation and
Pharmacological activation of AMPK and glucose uptake in cultured human skeletal muscle cells from patients with ME/CFS.
Brown AE et al. (2018) Pharmacological activation of AMPK and
Metabolomic Evidence for Peroxisomal Dysfunction in Myalgic Encephalomyelitis/Chronic Fatigue Syndrome.
Che X et al. (2022) Metabolomic Evidence for Peroxisomal Dysfunction
Bottom-up proteomics suggests an association between differential expression of mitochondrial proteins and chronic fatigue syndrome.
oCiregia F et al. (2016) Bottom-up proteomics suggests an association
Bioenergetic and Proteomic Profiling of Immune Cells in Myalgic Encephalomyelitis/Chronic Fatigue Syndrome Patients: An Exploratory Study.
Fernandez-Guerra P et al . (2021) Bioenergetic and Proteomic Profiling
A systematic review of mitochondrial abnormalities in myalgic encephalomyelitis/chronic fatigue syndrome/systemic exertion intolerance disease.
Holden S et al. (2020) A systematic review of mitochondrial