Investigating Immune Reactivity to the Intestinal Microbiome in Myalgic Encephalomyelitis/Chronic Fatigue Syndrome.
Seaton K (2022) Investigating Immune Reactivity to the Intestinal Microbiome […]
Research Papers
Myalgic Encephalomyelitis/Chronic Fatigue Syndrome as a Hyper-Regulated Immune System Driven by an Interplay Between Regulatory T Cells and Chronic Human Herpesvirus Infections.
Sepulveda N et al. (2019) Myalgic Encephalomyelitis/Chronic Fatigue Syndrome as
Fatigue and sarcoidosis.
Fatigue and sarcoidosis. Read More »
Sharma O . (1999) Fatigue and sarcoidosis. European Respiratory Journal
Mitochondrial alterations in NK lymphocytes from ME/CFS patients.
Mitochondrial alterations in NK lymphocytes from ME/CFS patients. Read More »
Silvestre I et al. (2019) Mitochondrial alterations in NK lymphocytes
Humoral Immunity Profiling of Subjects with Myalgic Encephalomyelitis Using a Random Peptide Microarray Differentiates Cases from Controls with High Specificity and Sensitivity.
Singh S et al. (2018) Humoral Immunity Profiling of Subjects
Myalgic Encephalomyelitis/Chronic Fatigue Syndrome – Evidence for an autoimmune disease.
Myalgic Encephalomyelitis/Chronic Fatigue Syndrome – Evidence for an autoimmune disease. Read More »
Sotzny F et al. (2018) Myalgic Encephalomyelitis/Chronic Fatigue Syndrome –
Allergy and the chronic fatigue syndrome.
Allergy and the chronic fatigue syndrome. Read More »
Straus SE et al. (1988) Allergy and the chronic fatigue
Inflammatory proteins are altered in chronic fatigue syndrome – a systematic review and meta-analysis.
Strawbridge R et al. (2019) Inflammatory proteins are altered in
Daily cytokines fluctuations, driven by leptin, are associated with fatigue severity in chronic fatigue syndrome: evidence of inflammatory pathology.
Stringer EA et al. (2013) Daily cytokines fluctuations, driven by
Changes in the transcriptome of circulating immune cells of a New Zealand cohort with myalgic encephalomyelitis/chronic fatigue syndrome.
Sweetman E et al. (2019) Changes in the transcriptome of
Delineating the Association Between Soluble CD26 and Autoantibodies Against G-Protein Coupled Receptors, Immunological and Cardiovascular Parameters Identifies Distinct Patterns in Post-Infectious vs. Non-Infection-Triggered Myalgic Encephalomyelitis/Chronic Fatigue Syndrome.
Szklarski M et al. (2021) Delineating the Association Between Soluble
Unperturbed Cytotoxic Lymphocyte Phenotype and Function in Myalgic Encephalomyelitis/Chronic Fatigue Syndrome Patients.
Theorell J et al. (2017) Unperturbed Cytotoxic Lymphocyte Phenotype and
Myalgic Encephalomyelitis/Chronic Fatigue Syndrome: Efficacy of Repeat Immunoadsorption.
Myalgic Encephalomyelitis/Chronic Fatigue Syndrome: Efficacy of Repeat Immunoadsorption. Read More »
Tolle M et al. (2020) Myalgic Encephalomyelitis/Chronic Fatigue Syndrome: Efficacy
No replication of previously reported association with genetic variants in the T cell receptor alpha (TRA) locus for myalgic encephalomyelitis/chronic fatigue syndrome (ME/CFS).
Ueland M et al. (2022) No replication of previously reported
C-Reactive Protein Response in Patients with Post-Treatment Lyme Disease Symptoms versus those with Myalgic Encephalomyelitis/Chronic Fatigue Syndrome.
Uhde M et al. (2018) C-Reactive Protein Response in Patients
Neuroinflammation and cytokines in myalgic encephalomyelitis/ chronic fatigue syndrome (ME/CFS): A critical review of research methods,
VanElzakker MB et al. (2018) Neuroinflammation and cytokines in myalgic
Systemic antibody responses against human microbiota flagellins are overrepresented in chronic fatigue syndrome patients.
Vogl T et al. (2022) Systemic antibody responses against human
Postinfective Fatigue Syndrome Is Not Associated with Altered Cytokine Production.
Postinfective Fatigue Syndrome Is Not Associated with Altered Cytokine Production. Read More »
Vollmer-Conna U et al. (2007) Postinfective Fatigue Syndrome Is Not
A systematic review of the association between fatigue and genetic polymorphisms.
A systematic review of the association between fatigue and genetic polymorphisms. Read More »
Wang T et al. (2017) A systematic review of the
A Unifying Hypothesis of the Pathophysiology of Myalgic Encephalomyelitis/Chronic Fatigue Syndrome (ME/CFS): Recognitions from the finding of autoantibodies against 2-adrenergic receptors.
Wirth K and Scheibenbogen C (2020) A Unifying Hypothesis of